Memory and Recall
Neuron and Nerve Impulse.
AMPA Receptors Determine Synaptic Strength.
NMDA Receptors Control Memory Processes.
Long-Term Potentiation and Depression.
The Role of CaMKII in Synaptic Plasticity.
Memory Engram Cells Could be Spiny Neurons.
NMDA Spikes and Plateaus are Required for Exciting Memory Engram Cells.
The Single Dendritic Branch as a Fundamental Memory Unit.
Born to Forget: Silent Neurons in the Dentate Gyrus.
Activation of Silent Neurons by Reminding Cues.
The Extinction (Desensitized) State of NMDA Receptors.
NMDAR Extinction Is Regulated by Protein Kinase A.
NMDAR Extinction in Dendritic Spines.
NMDAR Extinction in the Dendritic Shaft.
NMDAR Extinction: The Origin of Short Term Potentiation.
Why Can't We Recall Early Childhood Memories?
Regulation of both LTP and LTD by Ca
The General Mechanism of Memory Extinction and Retrieval.
Regulation of CABT-GluN2B Binding by Actin Depolymerization
How Shank3 Deficiency Causes NMDAR Hypofunction.
Memory Extinction: The Role of BDNF.
Memory Extinction: The Role of Dopamine.
Memory Retrieval: The Role of Norepinephrine.
Memory Retrieval: The Role of Acetylcholine.
Memory Consolidation: Formation of Enduring Memory.
Memory Consolidation: CRMP2 Directs Neurite Growth.
Memory Consolidation: Filopodia Guide Axonal Branching.
Memory Consolidation: The Role of PKMζ.
Memory Consolidation and Drug Addiction.
Memory Consolidation: The Memory Engram Network.
Memory Consolidation: Sharp Wave Ripples Provide Glutamate.
Memory Consolidation: Slow Oscillations Are Paced by Locus Coeruleus.
Memory Consolidation: Astrocytes Regulate the UP State.
Memory Consolidation: NMDAR Extinction may Cause the DOWN State.
in Memory Consolidation, Erasure, Extinction and Retrieval.
Born to Forget: The Underlying Mechanism.
Where is Working Memory Located?