Born to Forget, Die to Remember
Neuron and Nerve Impulse.
AMPA Receptors Determine Synaptic Strength.
NMDA Receptors Control Memory Processes.
Long-Term Potentiation and Depression.
The Role of CaMKII in Synaptic Plasticity.
Memory Engram Cells and Spiny Neurons.
Born to Forget: Silent Neurons in Dentate Gyrus.
Activation of Silent Neurons by Reminding Cues.
Memory Units, NMDA Spike and Plateau.
The Extinction State of NMDA Receptors.
Why Can't We Recall Early Childhood Memories?
Tubulin Inhibition Is Regulated by Protein Kinase A and Calcineurin.
Tubulin Inhibition of NMDA Receptors in Spines.
Tubulin Inhibition of NMDA Receptors in Dendritic Shaft.
Tubulin Inhibition May Cause Decline of Short Term Potentiation.
The General Mechanism of Memory Extinction and Retrieval.
Does It All: LTP, LTD, Extinction and Retrieval.
Memory Extinction: The Role of BDNF.
Memory Extinction: The Role of Dopamine.
Memory Retrieval: The Role of Norepinephrine.
Memory Retrieval: The Role of Acetylcholine.
Memory Consolidation: Generation of Sharp Wave Ripples in the Hippocampus.
Memory Consolidation: Generation of Slow Oscillations.
Memory Consolidation: The "Tags" of Newly Potentiated Memory Units.
Memory Consolidation: The Role of PKMζ.
Memory Consolidation: The Engram of Very Long-Term Memory.
Memory Consolidation: Tubulin Acetylation and Detyrosination.
Memory Consolidation: GluN2B-GluN2A Switch.
Memory Consolidation: Spine Generation and Elimination.
Memory Consolidation: Neurite Growth and Retraction
Memory Consolidation: Neurogenesis in Dentate Gyrus
Die to Remember: Tubulin Exodus from NMDARs in Dentate Gyrus.
Die to Remember: Evidence from End-of-Life EEG Surge.
Unlinked chapters are coming soon.